Read e-book online Management of Benign Biliary Stenosis and Injury: A PDF

By Elijah Dixon, Charles M. Vollmer Jr., Gary R. May

ISBN-10: 3319222724

ISBN-13: 9783319222721

ISBN-10: 3319222732

ISBN-13: 9783319222738

This quantity covers the pathophysiology, epidemiology, presentation, analysis, scientific and surgical administration of benign biliary stenosis. The e-book is uniquely established in a manner that enables parts of controversy to be highlighted by using a bankruptcy layout for every subject. every one bankruptcy subject is written through a professional within the box, with a moment professional highlighting controversies and delivering opposing viewpoints and therapy paradigms via a counterpoint bankruptcy instantly following the first bankruptcy. the quantity additionally gains an array of diagrams and illustrations.

As an authoritative textual content at the medical care of sufferers with benign biliary stenosis, Management of Benign Biliary Stenosis and Injury: A finished Guide is a precious source for all practitioners eager about the care of those sufferers, together with gastroenterologists, gastrointestinal surgeons, surgical oncologists, and transplant physicians.​

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Additional resources for Management of Benign Biliary Stenosis and Injury: A Comprehensive Guide

Sample text

MDR-related protein-2 (MRP2) has also been shown to transport certain bile salts into the canaliculi, along with the export of other organic solutes, including conjugated bilirubin, chemotherapeutic agents, antibiotics, toxins, and heavy metals [76, 77]. 1 Biliary System Anatomy, Physiology, and Embryology 27 Biliary Lipid and Cholesterol Secretion The secretion of phospholipids involves the translocation of phosphatidylcholine from the inner to the outer leaflet of the canalicular plasma membrane, which is mediated by the MDR3 transporter.

The two primary bile acids are synthesized from cholesterol by hepatocytes via two main pathways. The classic pathway, the primary mode of bile acid synthesis, leads to the formation of cholic acid, which constitutes the vast majority of the bile acid pool. The alternate pathway leads to the formation of chenodeoxycholic acid. Once secreted into the lumen of the intestine, a small percentage of the cholic and chenodeoxycholic acids are dehydroxylated by intestinal bacteria, producing the two secondary bile acids: deoxycholic acid and lithocholic acid, respectively [68, 69].

Hentsch B, Lyons I, Li R, Hartley L, Lints TJ, Adams JM, et al. Hlx homeo box gene is essential for an inductive tissue interaction that drives expansion of embryonic liver and gut. Genes Dev. 1996;10(1):70–9. PubMed. 1 Biliary System Anatomy, Physiology, and Embryology 35 20. Weinstein M, Monga SP, Liu Y, Brodie SG, Tang Y, Li C, et al. Smad proteins and hepatocyte growth factor control parallel regulatory pathways that converge on beta1-integrin to promote normal liver development. Mol Cell Biol.

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Management of Benign Biliary Stenosis and Injury: A Comprehensive Guide by Elijah Dixon, Charles M. Vollmer Jr., Gary R. May

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